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datadream

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  1. I really loved Sprawl. The whole intersecting plotlines (well multiple plot lines tying in only at the very end) makes things interesting for me. Except when I'm not into the book itself. If I'm not into a book then that style just pisses me off enough to burn the books.

     

    No offense to Robert Jordan...

     

    Okay no one mentioned Jordan but honestly I find him too wordy and something about this reply reminds me of him.

     

    I'm actually thinking about reading more from the author of Snow crash. Actually never read Snow crash. Is it any good? I have all his works on my Kindle but haven't been reading much lately.

  2. So your character pops dex tabs too much in game and you really want to know how you can round out character, make them more realistic. Well in a Cyberpunk world the best way of doing this is by giving your character some flaws. What a better way to do this than suggest possible neurological disorders? :huh:

     

    Disorders & The Mind is my post that deals with both neurological disorders present in the Cyberpunk world and various thematic elements present in Cyberpunk writings that deal with the mind. While by the sheer nature of this post such an issue can be portrayed in an exhaustive way, I'll try to highlight such elements without boring you.

     

    Since Cyberpunk primarily deals with technology and chemicals that effect the brain, a number of neurological conditions might exist. While all characters aren't prone to disease just because of recreational drug use, these elements - at your discretion - might be used to give your character some depth. I've listed a number of different types of diseases here which can be searched by using the "Find" feature on your browser and entering various strings.

     

    For instance if you have a character that is very physical and uses technology that boosts ATP in muscles then perhaps that character also experiences problems related to ATP production (namely just about every muscular disease out there). But don't be limited to real world disorders either, combine and match whatever you think the next advancement in medicine and technology would be. Plenty of sci-fi authors "expand" the truth a bit when creating disorders for their characters.

     

    Keep in mind that some of these disorders can be birth defects. You should probably do some research on them if you decide to use them. Whether your character knows he or she has such a disease in character is up to you (just keep it realistic).

     

     

    To create this list I've visited a number of websites. Most namely the National Institute of Neurological Disorders and Stroke, Wikipedia, various encyclopedias, the manual for psychiatric diagnosis and a few other sources. If you have RP books that contain useful neurological disorders that you'd like to see on this list feel free to contact me with the related information.

     

    Hope this list helps!

     

    D

     

    ******

    Terms to Know:

     

    CNS - Central Nervous System. Composed of your brain, spinal cord and retina.

    CSF - Cerebrospinal fluid.

    GABA - gamma-Aminobutyric acid; main inhibitory neurotransmitter.

    Receptor site - Neurotransmitter receptors.

    Synapse -junction that permits a neuron to pass an electrical or chemical signal to another cell. TWO types exist - chemical which is released when presynaptic neuron fires a neurotransmitter that connects to a receptor site (in the postsynaptic cell). And electrical or the channels that are between such presynaptic and postsynaptic cells and allow the passage of electrical current.

    Neuroscience - the study of the nervous system.

     

    Neurons and synapses - Wikipedia puts it best (in easy terms):

     

    Most neurons send signals via their axons, although some types are capable of dendrite-to-dendrite communication. (In fact, the types of neurons called amacrine cells have no axons, and communicate only via their dendrites.) Neural signals propagate along an axon in the form of electrochemical waves called action potentials, which produce cell-to-cell signals at points where axon terminals make synaptic contact with other cells.[32].

     

    Synapses may be electrical or chemical. Electrical synapses make direct electrical connections between neurons,[33] but chemical synapses are much more common, and much more diverse in function.[34] At a chemical synapse, the cell that sends signals is called presynaptic, and the cell that receives signals is called postsynaptic. Both the presynaptic and postsynaptic areas are full of molecular machinery that carries out the signalling process. The presynaptic area contains large numbers of tiny spherical vessels called synaptic vesicles, packed with neurotransmitter chemicals.[32] When the presynaptic terminal is electrically stimulated, an array of molecules embedded in the membrane are activated, and cause the contents of the vesicles to be released into the narrow space between the presynaptic and postsynaptic membranes, called the synaptic cleft. The neurotransmitter then binds to receptors embedded in the postsynaptic membrane, causing them to enter an activated state.[34] Depending on the type of receptor, the resulting effect on the postsynaptic cell may be excitatory, inhibitory, or modulatory in more complex ways. For example, release of the neurotransmitter acetylcholine at a synaptic contact between a motor neuron and a muscle cell induces rapid contraction of the muscle cell.[35] The entire synaptic transmission process takes only a fraction of a millisecond, although the effects on the postsynaptic cell may last much longer (even indefinitely, in cases where the synaptic signal leads to the formation of a memory trace).[8]

    Structure of a typical chemical synapse

    Synapse Illustration unlabeled.

     

    There are literally hundreds of different types of synapses. In fact, there are over a hundred known neurotransmitters, and many of them have multiple types of receptor.[36] Many synapses use more than one neurotransmitter—a common arrangement is for a synapse to use one fast-acting small-molecule neurotransmitter such as glutamate or GABA, along with one or more peptide neurotransmitters that play slower-acting modulatory roles. Molecular neuroscientists generally divide receptors into two broad groups: chemically gated ion channels and second messenger systems. When a chemically gated ion channel is activated, it forms a passage that allow specific types of ion to flow across the membrane. Depending on the type of ion, the effect on the target cell may be excitatory or inhibitory. When a second messenger system is activated, it starts a cascade of molecular interactions inside the target cell, which may ultimately produce a wide variety of complex effects, such as increasing or decreasing the sensitivity of the cell to stimuli, or even altering gene transcription.

     

    According to a rule called Dale's principle, which has only a few known exceptions, a neuron releases the same neurotransmitters at all of its synapses.[37] This does not mean, though, that a neuron exerts the same effect on all of its targets, because the effect of a synapse depends not on the neurotransmitter, but on the receptors that it activates.[34] Because different targets can (and frequently do) use different types of receptors, it is possible for a neuron to have excitatory effects on one set of target cells, inhibitory effects on others, and complex modulatory effects on others still. Nevertheless, it happens that the two most widely used neurotransmitters, glutamate and GABA, each have largely consistent effects. Glutamate has several widely occurring types of receptors, but all of them are excitatory or modulatory. Similarly, GABA has several widely occurring receptor types, but all of them are inhibitory.[38] Because of this consistency, glutamatergic cells are frequently referred to as "excitatory neurons", and GABAergic cells as "inhibitory neurons". Strictly speaking this is an abuse of terminology—it is the receptors that are excitatory and inhibitory, not the neurons—but it is commonly seen even in scholarly publications.

     

    One very important subset of synapses are capable of forming memory traces by means of long-lasting activity-dependent changes in synaptic strength.[39] The best-known form of neural memory is a process called long-term potential (abbreviated LTP), which operates at synapses that use the neurotransmitter glutamate acting on a special type of receptor known as the NMDA receptor.[40] The NMDA receptor has an "associative" property: if the two cells involved in the synapse are both activated at approximately the same time, a channel opens that permits calcium to flow into the target cell.[41] The calcium entry initiates a second messenger cascade that ultimately leads to an increase in the number of glutamate receptors in the target cell, thereby increasing the effective strength of the synapse. This change in strength can last for weeks or longer. Since the discovery of LTP in 1973, many other types of synaptic memory traces have been found, involving increases or decreases in synaptic strength that are induced by varying conditions, and last for variable periods of time.[40] Reward learning, for example, depends on a variant form of LTP that is conditioned on an extra input coming from a reward-signalling pathway that uses dopamine as neurotransmitter.[42] All these forms of synaptic modifiability, taken collectively, give rise to neural plasticity, that is, to a capability for the nervous system to adapt itself to variations in the environment.

     

    Neurological Diseases by Terms:

     

    A

     

    Absence of the Septum Pellucidum

    Acid Lipase Disease

    Acid Maltase Deficiency

    Acquired Epileptiform Aphasia

    Acute Disseminated Encephalomyelitis

     

    ADHD

    Adie's Pupil

    Adie's Syndrome

    Adrenoleukodystrophy

    Agenesis of the Corpus Callosum

     

    Agnosia

    Aicardi Syndrome

    Aicardi-Goutieres Syndrome Disorder

    AIDS - Neurological Complications

    Alexander Disease

     

    Alpers' Disease

    Alternating Hemiplegia

    Alzheimer's Disease

    Amyotrophic Lateral Sclerosis

    Anencephaly

     

    Aneurysm

    Angelman Syndrome

    Angiomatosis

    Anoxia

    Antiphospholipid Syndrome

     

    Aphasia

    Apraxia

    Arachnoid Cysts

    Arachnoiditis

    Arnold-Chiari Malformation

     

    Arteriovenous Malformation

    Asperger Syndrome

    Ataxia

    Ataxia Telangiectasia

    Ataxias and Cerebellar or Spinocerebellar Degeneration

     

    Atrial Fibrillation and Stroke

    Attention Deficit-Hyperactivity Disorder

    Autism

    Autonomic Dysfunction

     

    B

     

    Barth Syndrome

    Batten Disease

    Becker's Myotonia

    Behcet's Disease

     

    Bell's Palsy

    Benign Essential Blepharospasm

    Benign Focal Amyotrophy

    Benign Intracranial Hypertension

    Bernhardt-Roth Syndrome

     

    Binswanger's Disease

    Blepharospasm

    Bloch-Sulzberger Syndrome

    Brachial Plexus Birth Injuries

    Brachial Plexus Injuries

     

    Bradbury-Eggleston Syndrome

    Brain and Spinal Tumors

    Brain Aneurysm

    Brain Injury

    Brown-Sequard Syndrome

     

    Bulbospinal Muscular Atrophy

     

    C

     

    CADASIL

    Canavan Disease

    Carpal Tunnel Syndrome

    Causalgia

    Cavernomas

     

    Cavernous Angioma

    Cavernous Malformation

    Central Cervical Cord Syndrome

    Central Cord Syndrome

    Central Pain Syndrome

     

    Central Pontine Myelinolysis

    Cephalic Disorders

    Ceramidase Deficiency

    Cerebellar Degeneration

    Cerebellar Hypoplasia

     

    Cerebral Aneurysm

    Cerebral Arteriosclerosis

    Cerebral Atrophy

    Cerebral Beriberi

    Cerebral Cavernous Malformation

     

    Cerebral Gigantism

    Cerebral Hypoxia

    Cerebral Palsy

    Cerebro-Oculo-Facio-Skeletal Syndrome

    Charcot-Marie-Tooth Disease

     

    Chiari Malformation

    Cholesterol Ester Storage Disease

    Chorea

    Choreoacanthocytosis

    Chronic Inflammatory Demyelinating Polyneuropathy (CIDP)

     

    Chronic Orthostatic Intolerance

    Chronic Pain

    Cockayne Syndrome Type II

    Coffin Lowry Syndrome

    COFS

     

    Colpocephaly

    Coma

    Complex Regional Pain Syndrome

    Congenital Facial Diplegia

    Congenital Myasthenia

     

    Congenital Myopathy

    Congenital Vascular Cavernous Malformations

    Corticobasal Degeneration

    Cranial Arteritis

    Craniosynostosis

     

    Creutzfeldt-Jakob Disease

    Cumulative Trauma Disorders

    Cushing's Syndrome

    Cytomegalic Inclusion Body Disease

    Cytomegalovirus Infection

     

    D

     

    Dancing Eyes-Dancing Feet Syndrome

    Dandy-Walker Syndrome

    Dawson Disease

    De Morsier's Syndrome

    Deep Brain Stimulation for Parkinson's Disease

     

    Dejerine-Klumpke Palsy

    Dementia - Multi-infarct, semantic, subcortical

     

    Dementia With Lewy Bodies

    Dentate Cerebellar Ataxia

    Dentatorubral Atrophy

    Dermatomyositis

    Developmental Dyspraxia

     

    Devic's Syndrome

    Diabetic Neuropathy

    Diffuse Sclerosis

    Dravet Syndrome

    Dysautonomia

     

    Dysgraphia

    Dyslexia

    Dysphagia

    Dyspraxia

    Dyssynergia Cerebellaris Myoclonica

     

    Dyssynergia Cerebellaris Progressiva

    Dystonias

     

    E

     

    Empty Sella Syndrome

    Encephalitis

    Encephalitis Lethargica

    Encephaloceles

     

    Encephalopathy

    Encephalopathy, familial infantile, with intracranial calcification and chronic cerebrospinal fluid lymphocytosis; Cree encephalitis; Pseudo-Torch syndrome; Pseudotoxoplasmosis syndrome

    Encephalotrigeminal Angiomatosis

    Epilepsy

    Epileptic Hemiplegia

     

    Erb-Duchenne and Dejerine-Klumpke Palsies

    Erb's Palsy

    Essential Tremor

    Extrapontine Myelinolysis

     

    F

     

    Fabry Disease

    Fahr's Syndrome

    Fainting

    Familial Dysautonomia

    Familial Hemangioma

     

    Familial Idiopathic Basal Ganglia Calcification

    Familial Periodic Paralyses

    Familial Spastic Paralysis

    Farber's Disease

    Febrile Seizures

     

    Fibromuscular Dysplasia

    Fisher Syndrome

    Floppy Infant Syndrome

    Foot Drop

    Friedreich's Ataxia

     

    Frontotemporal Dementia

     

    G

     

    Gangliosidoses

    Gaucher's Disease

    Gerstmann's Syndrome

    Gerstmann-Straussler-Scheinker Disease

    Giant Axonal Neuropathy

     

    Giant Cell Arteritis

    Giant Cell Inclusion Disease

    Globoid Cell Leukodystrophy

    Glossopharyngeal Neuralgia

    Glycogen Storage Disease

     

    Guillain-Barré Syndrome

     

    H

     

    Hallervorden-Spatz Disease

    Head Injury

    Headache

    Hemicrania Continua

    Hemifacial Spasm

     

    Hemiplegia Alterans

    Hereditary Neuropathies

    Hereditary Spastic Paraplegia

    Heredopathia Atactica Polyneuritiformis

    Herpes Zoster

     

    Herpes Zoster Oticus

    Hirayama Syndrome

    Holmes-Adie syndrome

    Holoprosencephaly

    HTLV-1 Associated Myelopathy

     

    Hughes Syndrome

    Huntington's Disease

    Hydranencephaly

    Hydrocephalus

    Hydrocephalus - Normal Pressure

     

    Hydromyelia

    Hypercortisolism

    Hypersomnia

    Hypertonia

    Hypotonia

     

    Hypoxia

     

    I

     

    Immune-Mediated Encephalomyelitis

    Inclusion Body Myositis

    Incontinentia Pigmenti

    Infantile Hypotonia

    Infantile Neuroaxonal Dystrophy

     

    Infantile Phytanic Acid Storage Disease

    Infantile Refsum Disease

    Infantile Spasms

    Inflammatory Myopathies

    Iniencephaly

     

    Intestinal Lipodystrophy

    Intracranial Cysts

    Intracranial Hypertension

    Isaac's Syndrome

     

    J

     

    Joubert Syndrome

     

    K

     

    Kearns-Sayre Syndrome

    Kennedy's Disease

    Kinsbourne syndrome

    Kleine-Levin Syndrome

    Klippel-Feil Syndrome

     

    Klippel-Trenaunay Syndrome (KTS)

    Klüver-Bucy Syndrome

    Korsakoff's Amnesic Syndrome

    Krabbe Disease

    Kugelberg-Welander Disease

     

    Kuru

     

    L

     

    Lambert-Eaton Myasthenic Syndrome

    Landau-Kleffner Syndrome

    Lateral Femoral Cutaneous Nerve Entrapment

    Lateral Medullary Syndrome

    Learning Disabilities

     

    Leigh's Disease

    Lennox-Gastaut Syndrome

    Lesch-Nyhan Syndrome

    Leukodystrophy

    Levine-Critchley Syndrome

     

    Lewy Body Dementia

    Lipid Storage Diseases

    Lipoid Proteinosis

    Lissencephaly

    Locked-In Syndrome

     

    Lou Gehrig's Disease

    Lupus - Neurological Sequelae

    Lyme Disease - Neurological Complications

     

    M

     

    Machado-Joseph Disease

    Macrencephaly

    Megalencephaly

    Melkersson-Rosenthal Syndrome

    Meningitis

     

    Meningitis and Encephalitis

    Menkes Disease

    Meralgia Paresthetica

    Metachromatic Leukodystrophy

    Microcephaly

     

    Migraine

    Miller Fisher Syndrome

    Mini-Strokes

    Mitochondrial Myopathies

    Moebius Syndrome

     

    Monomelic Amyotrophy

    Motor Neuron Diseases

    Moyamoya Disease

    Mucolipidoses

    Mucopolysaccharidoses

     

    Multifocal Motor Neuropathy

    Multi-Infarct Dementia

    Multiple Sclerosis

    Multiple System Atrophy

    Multiple System Atrophy with Orthostatic Hypotension

     

    Muscular Dystrophy

    Myasthenia - Congenital

    Myasthenia Gravis

    Myelinoclastic Diffuse Sclerosis

    Myoclonic Encephalopathy of Infants

     

    Myoclonus

    Myopathy

    Myopathy - Congenital

    Myopathy - Thyrotoxic

    Myotonia

     

    Myotonia Congenita

     

    N

     

    Narcolepsy

    Neuroacanthocytosis

    Neurodegeneration with Brain Iron Accumulation

    Neurofibromatosis

    Neuroleptic Malignant Syndrome

     

    Neurological Complications of AIDS

    Neurological Complications Of Lyme Disease

    Neurological Consequences of Cytomegalovirus Infection

    Neurological Manifestations of Pompe Disease

    Neurological Sequelae Of Lupus

     

    Neuromyelitis Optica

    Neuromyotonia

    Neuronal Ceroid Lipofuscinosis

    Neuronal Migration Disorders

    Neuropathy - Hereditary

     

    Neurosarcoidosis

    Neurosyphilis

    Neurotoxicity

    Nevus Cavernosus

    Niemann-Pick Disease

     

    Normal Pressure Hydrocephalus

     

    O

     

    Occipital Neuralgia

    Ohtahara Syndrome

    Olivopontocerebellar Atrophy

    Opsoclonus Myoclonus

    Orthostatic Hypotension

     

    O'Sullivan-McLeod Syndrome

    Overuse Syndrome

     

    P

     

    Pain - Chronic

    Pantothenate Kinase-Associated Neurodegeneration

    Paraneoplastic Syndromes

    Paresthesia

    Parkinson's Disease

     

    Paroxysmal Choreoathetosis

    Paroxysmal Hemicrania

    Parry-Romberg

    Pelizaeus-Merzbacher Disease

    Pena Shokeir II Syndrome

     

    Perineural Cysts

    Periodic Paralyses

    Peripheral Neuropathy

    Periventricular Leukomalacia

    Persistent Vegetative State

     

    Pervasive Developmental Disorders

    Phytanic Acid Storage Disease

    Pick's Disease

    Pinched Nerve

    Piriformis Syndrome

     

    Pituitary Tumors

    Polymyositis

    Pompe Disease

    Porencephaly

    Postherpetic Neuralgia

     

    Postinfectious Encephalomyelitis

    Post-Polio Syndrome

    Postural Hypotension

    Postural Orthostatic Tachycardia Syndrome

    Postural Tachycardia Syndrome

     

    Primary Dentatum Atrophy

    Primary Lateral Sclerosis

    Primary Progressive Aphasia

    Prion Diseases

    Progressive Hemifacial Atrophy

     

    Progressive Locomotor Ataxia

    Progressive Multifocal Leukoencephalopathy

    Progressive Sclerosing Poliodystrophy

    Progressive Supranuclear Palsy

    Prosopagnosia

     

    Pseudotumor Cerebri

     

    Q

     

    None.

     

    R

     

    Ramsay Hunt Syndrome II

    Rasmussen's Encephalitis

    Reflex Sympathetic Dystrophy Syndrome

    Refsum Disease

     

    Refsum Disease - Infantile

    Repetitive Motion Disorders

    Repetitive Stress Injuries

    Restless Legs Syndrome

    Retrovirus-Associated Myelopathy

     

    Rett Syndrome

    Reye's Syndrome

    Rheumatic Encephalitis

    Riley-Day Syndrome

     

    S

     

    Sacral Nerve Root Cysts

    Saint Vitus Dance

    Salivary Gland Disease

    Sandhoff Disease

    Schilder's Disease

     

    Schizencephaly

    Seitelberger Disease

    Seizure Disorder

    Semantic Dementia

    Septo-Optic Dysplasia

     

    Severe Myoclonic Epilepsy of Infancy (SMEI)

    Shaken Baby Syndrome

    Shingles

    Shy-Drager Syndrome

    Sjögren's Syndrome

     

    Sleep Apnea

    Sleeping Sickness

    Sotos Syndrome

    Spasticity

    Spina Bifida

     

    Spinal Cord Infarction

    Spinal Cord Injury

    Spinal Cord Tumors

    Spinal Muscular Atrophy

    Spinocerebellar Atrophy

     

    Spinocerebellar Degeneration

    Steele-Richardson-Olszewski Syndrome

    Stiff-Person Syndrome

    Striatonigral Degeneration

    Stroke

     

    Sturge-Weber Syndrome

    Subacute Sclerosing Panencephalitis

    Subcortical Arteriosclerotic Encephalopathy

    SUNCT Headache

    Swallowing Disorders

     

    Sydenham Chorea

    Syncope

    Syphilitic Spinal Sclerosis

    Syringohydromyelia

    Syringomyelia

     

    Systemic Lupus Erythematosus

     

    T

     

    Tabes Dorsalis

    Tardive Dyskinesia

    Tarlov Cysts

    Tay-Sachs Disease

    Temporal Arteritis

     

    Tethered Spinal Cord Syndrome

    Thomsen's Myotonia

    Thoracic Outlet Syndrome

    Thyrotoxic Myopathy

    Tic Douloureux

     

    Todd's Paralysis

    Tourette Syndrome

    Transient Ischemic Attack

    Transmissible Spongiform Encephalopathies

    Transverse Myelitis

     

    Traumatic Brain Injury

    Tremor

    Trigeminal Neuralgia

    Tropical Spastic Paraparesis

    Troyer Syndrome

     

    Tuberous Sclerosis

     

    U

     

    None.

     

    V

     

    Vascular Erectile Tumor

    Vasculitis Syndromes of the Central and Peripheral Nervous Systems

    Von Economo’s Disease

    Von Hippel-Lindau Disease (VHL)

    Von Recklinghausen's Disease

     

    W

     

    Wallenberg's Syndrome

    Werdnig-Hoffman Disease

    Wernicke-Korsakoff Syndrome

    West Syndrome

    Whiplash

     

    Whipple's Disease

    Williams Syndrome

    Wilson's Disease

    Wolman’s Disease

     

    X

     

    X-Linked Spinal and Bulbar Muscular Atrophy

     

    Y

     

    None.

     

    Z

     

    Zellweger Syndrome

     

    Noted:

     

    Overuse Syndrome - Repetitive motion disorders (RMDs) are a family of muscular conditions that result from repeated motions performed in the course of normal work or daily activities. RMDs include carpal tunnel syndrome, bursitis, tendonitis, epicondylitis, ganglion cyst, tenosynovitis, and trigger finger. RMDs are caused by too many uninterrupted repetitions of an activity or motion, unnatural or awkward motions such as twisting the arm or wrist, overexertion, incorrect posture, or muscle fatigue. RMDs occur most commonly in the hands, wrists, elbows, and shoulders, but can also happen in the neck, back, hips, knees, feet, legs, and ankles. The disorders are characterized by pain, tingling, numbness, visible swelling or redness of the affected area, and the loss of flexibility and strength. For some individuals, there may be no visible sign of injury, although they may find it hard to perform easy tasks Over time, RMDs can cause temporary or permanent damage to the soft tissues in the body -- such as the muscles, nerves, tendons, and ligaments - and compression of nerves or tissue. Generally, RMDs affect individuals who perform repetitive tasks such as assembly line work, meatpacking, sewing, playing musical instruments, and computer work.

     

    Holmes-Adie syndrome - is a neurological disorder affecting the pupil of the eye and the autonomic nervous system. It is characterized by one eye with a pupil that is larger than normal and constricts slowly in bright light (tonic pupil), along with the absence of deep tendon reflexes, usually in the Achilles tendon. HAS is thought to be the result of a viral or bacterial infection that causes inflammation and damage to neurons in the ciliary ganglion, an area of the brain that controls eye movements, and the spinal ganglion, an area of the brain involved in the response of the autonomic nervous system.

     

    Adrenoleukodystrophy (ALD) - is one of a group of genetic disorders called the leukodystrophies that cause damage to the myelin sheath, an insulating membrane that surrounds nerve cells in the brain. People with ALD accumulate high levels of saturated, very long chain fatty acids (VLCFA) in the brain and adrenal cortex because they do not produce the enzyme that breaks down these fatty acids in the normal manner. The loss of myelin and the progressive dysfunction of the adrenal gland are the primary characteristics of ALD. ALD has two subtypes. The most common is the X-linked form (X-ALD), which involves an abnormal gene located on the X-chromosome. Women have two X-chromosomes and are the carriers of the disease, but since men only have one X-chromosome and lack the protective effect of the extra X-chromosome, they are more severely affected. Onset of X-ALD can occur in childhood or in adulthood. The childhood form is the most severe, with onset between ages 4 and 10. The most common symptoms are usually behavioral changes such as abnormal withdrawal or aggression, poor memory, and poor school performance. Other symptoms include visual loss, learning disabilities, seizures, poorly articulated speech, difficulty swallowing, deafness, disturbances of gait and coordination, fatigue, intermittent vomiting, increased skin pigmentation, and progressive dementia.

     

    Cerebral aneurysm - is the dilation, bulging, or ballooning-out of part of the wall of an artery in the brain. Cerebral aneurysms can occur at any age, although they are more common in adults than in children and are slightly more common in women than in men.

     

    Apraxia (more severe form of dyspraxia) - is a neurological disorder characterized by loss of the ability to execute or carry out skilled movements and gestures, despite having the desire and the physical ability to perform them. Apraxia results from dysfunction of the cerebral hemispheres of the brain, especially the parietal lobe, and can arise from many diseases or damage to the brain.

     

    Autism - is characterized by three distinctive behaviors. Autistic children have difficulties with social interaction, display problems with verbal and nonverbal communication, and exhibit repetitive behaviors or narrow, obsessive interests. These behaviors can range in impact from mild to disabling. Autism varies widely in its severity and symptoms and may go unrecognized, especially in mildly affected children or when more debilitating handicaps mask it. Scientists aren’t certain what causes autism, but it’s likely that both genetics and environment play a role.

     

    Coma - sometimes also called persistent vegetative state, is a profound or deep state of unconsciousness. Persistent vegetative state is not brain-death. An individual in a state of coma is alive but unable to move or respond to his or her environment.

     

    Cushing's syndrome - also called hypercortisolism, is a rare endocrine disorder caused by chronic exposure of the body's tissues to excess levels of cortisol - a hormone naturally produced by the adrenal gland. Exposure to too much cortisol can occur from long-term use of synthetic glucocorticoid hormones to treat inflammatory illnesses. Pituitary adenomas (benign tumors of the pituitary gland) that secrete increased amounts of ACTH (adrenocorticotropic hormone, a substance that controls the release of cortisol) can also spur overproduction of cortisol. Tumors of the adrenal gland and ectopic ACTH syndrome (a condition in which ACTH is produced by various types of potentially malignant tumors that occur in different parts of the body) can cause similar problems with cortisol balance. Common symptoms of Cushing's syndrome include upper body obesity, severe fatigue and muscle weakness, high blood pressure, backache, elevated blood sugar, easy bruising, and bluish-red stretch marks on the skin. In women, there may be increased growth of facial and body hair, and menstrual periods may become irregular or stop completely. Neurological symptoms include difficulties with memory and neuromuscular disorders.

     

    Dementia - is not a specific disease. It is a descriptive term for a collection of symptoms that can be caused by a number of disorders that affect the brain. People with dementia have significantly impaired intellectual functioning that interferes with normal activities and relationships. They also lose their ability to solve problems and maintain emotional control, and they may experience personality changes and behavioral problems, such as agitation, delusions, and hallucinations.

     

    Hypersomnia - is characterized by recurrent episodes of excessive daytime sleepiness or prolonged nighttime sleep. Different from feeling tired due to lack of or interrupted sleep at night, persons with hypersomnia are compelled to nap repeatedly during the day, often at inappropriate times such as at work, during a meal, or in conversation.

     

    Cerebral hypoxia - refers to a condition in which there is a decrease of oxygen supply to the brain even though there is adequate blood flow. Drowning, strangling, choking, suffocation, cardiac arrest, head trauma, carbon monoxide poisoning, and complications of general anesthesia can create conditions that can lead to cerebral hypoxia.

     

     

    Related Things to Know:

     

    Cybernetics - A system of control theory and systems theory which deals with computers, psychology, sociology, biology and economics. This theory was very much established by genius Norbert Wiener, an American mathematician, and proponent of systems theory.

     

    Alonzo Church - The god father of theoretical computer science and father of lambda calculus.

     

    Lambda calculus - - this is a form of mathematics which deals with function definition, function application and recursion in attempts to understand the foundations of mathematics itself. Through this system of higher level mathematics he was able to pave the way for computer programmers (functional programming). This directly related to the development of the programming language Lisp (best known language behind the Emacs editor).

     

    Turing Machine - Invented by Alan Turing in 1937 this is a theoretical device that, according to a set of rules, can simulate human thought. Instead of realistic computers (as we have now), this was designed as a thought experiment to help scientists determine the limitations of mechanical computations and "machine thought." Why was this important? Because it also formed a basis for our own preconceived limitations of thought related to human beings. These concepts would become powerful in the world of computing and neurological studies.

     

    Turing Test - Made possible by mathematics presented by Church and concepts delivered by Alan Turing, this test - named after Turing - serves as a logical "proof" for a machine's given intelligence. Should a machine actually reply intuitively to one such test it would reach true "intelligence" independent of humans and, essentially, be separate from the human mind.

     

    Chinese Room - Probably the single most impressive experiment ever came from John Seasrle in the 1980s which disproved the concept of the Turing Test. He maintained - through real world testing - that just because something replies in an intelligent way consistently, and seems to have expanding thoughts on the subject (by way of learning), that doesn't mean it is human, independent or intelligent. Searle argues that machines cannot think and has a test to prove it.

     

    For instance, say in one room you have a computer with a great AI program, he's separated by a wall from an actual human. The human draws Chinese characters onto a card and passes it through the door to the other side, awaiting a response. A card is passed back with a longer message, seemingly a response. Satisfied the process is repeated and what appears to be a conversation is conducted. The man with his cards thinks he's having an intelligent conversation with a human being.

     

    Essentially the machine is passing the Turing test, the AI in the other room is incredibly strong. The man is completely convinced that on the other side of the door there is an actual Chinese speaker. But consider the fact that the man doesn't know Chinese. He's writing symbols off the top of his head and the machine is giving him back reasonable output. But the test concludes that because the man does not actually know Chinese neither can the machine.

     

    Searle then supposes that he is in a closed room and has a book with an English version of the computer program, along with sufficient paper, pencils, erasers and filing cabinets. Searle could receive Chinese characters through a slot in the door, process them according to the program's instructions, and produce Chinese characters as output. As the computer had passed the Turing test this way, it is fair, says Searle, to deduce that he would be able to do so as well, simply by running the program manually.

     

    There is no essential difference, Searle argues, between the role of the computer in either situation. Each entity is following a program in a step-by-step fashion which simulates intelligence. Moreover machines that appear to think do not have a mind at all.

     

    Peter Watts - Hard Sci-fi writer enjoyed by many Cyberpunk fans uses the Chinese Room in his book Blindsight in which he asserts that the same might be true of humans (See Blindsight by Peter Watts for more information).

     

    (A1) "Programs are formal (syntactic)."

     

    A program uses syntax to manipulate symbols and pays no attention to the semantics of the symbols. It knows where to put the symbols and how to move them around, but it doesn't know what they stand for or what they mean. For the program, the symbols are just physical objects like any others.

     

    (A2) "Minds have mental contents (semantics)."

     

    Unlike the symbols used by a program, our thoughts have meaning: they represent things and we know what it is they represent.

     

    (A3) "Syntax by itself is neither constitutive of nor sufficient for semantics."

     

    This is what the Chinese room argument is intended to prove: the Chinese room has syntax (because there is a man in there moving symbols around). The Chinese room has no semantics (because, according to Searle, there is no one or nothing in the room that understands what the symbols mean). Therefore, having syntax is not enough to generate semantics.

     

    Searle posits that these lead directly to this conclusion:

     

    (C1) Programs are neither constitutive of nor sufficient for minds.

     

    This should follow without controversy from the first three: Programs don't have semantics. Programs have only syntax, and syntax is insufficient for semantics. Every mind has semantics. Therefore programs are not minds.

     

    He also claims:

     

    (C2) Any other system capable of causing minds would have to have causal powers (at least) equivalent to those of brains.

     

    Brains must have something that causes a mind to exist. Science has yet to determine exactly what it is, but it must exist, because minds exist. Searle calls it "causal powers". "Causal powers" is whatever the brain uses to create a mind. If anything else can cause a mind to exist, it must have "equivalent causal powers". "Equivalent causal powers" is whatever else that could be used to make a mind.

     

    And from this he derives the further conclusions:

     

    (C3) Any artifact that produced mental phenomena, any artificial brain, would have to be able to duplicate the specific causal powers of brains, and it could not do that just by running a formal program.

     

    This follows from C1 and C2: Since no program can produce a mind, and "equivalent causal powers" produce minds, it follows that programs do not have "equivalent causal powers."

     

    (C4) The way that human brains actually produce mental phenomena cannot be solely by virtue of running a computer program.

     

    Since programs do not have "equivalent causal powers", "equivalent causal powers" produce minds, and brains produce minds, it follows that brains do not use programs to produce minds.

     

    Why Is Systems Theory

    Philosophy and Thought Experiments

    Important When Understanding Neurological Disorders:

     

    In the Cyberpunk setting it is extremely important to take into consideration various neurological disorders resulting from the use of technology. Simply put we can also make the argument that such disorders - simply by existing in a world of advanced neurological thought and understanding - are incredibly important to the avid Cyberpunk fan. Whether we're talking about disorders or interface implants, the mind and its workings play a large role in understanding larger issues in Cyberpunk work.

     

    The best types of Cyberpunk writing combines various issues relating to human psychology, biology and even larger issues of sociology and macro-economics. For that reason it is important to list them here for additional consideration. While such major themes might not make their way into an individual's every day RPing, it is important to touch upon them for stylistic and thematic purposes.

  3. So if I somehow merge my brain with my computer systems and become a sort of cyborg and spend all day posting would that classify me as a spam bot?

     

    I just stumbled upon this site looking for some ideas in decorating my office at home. I'm a long time fan of the idea of merging biological systems with electronic systems. I guess you can say I've had pretty EXTREME ideas and now have a place to discuss them.

    You sound a lot like me so welcome!! (If you didn't I wouldn't welcome you...?? Yeah that came out weird.)

     

  4. NEWS NEWS NEWS NEWS NEWS NEWS NEWS

     

    There is nothing new to report. What follows is the area where news will be reported as it breaks in game...

     

     

    EURASIA

    * The UK-Hong Kong index reports an all time high for BBC-Viacom, hitting the 95.5 per share mark.

    * Market prices are on the rise for Eurasian business owners, CFO Benson Larsdale of BBC-Viacom Reports: “Stock value in all telecommunications stock is on the rise. Now is a great time to invest, BBC-Viacom is offering a superior dividends to all share holders with voting rights. Despite rumors to the contrary the industry is at an all time high. Consumer confidence is terrific.”

    * 98% of all stock transactions for BBC-Viacom – our parent company – have been traded by company's board members for the past 3 years. When asked if this represents corporate price inflation in attempts to boast the consumer's confidence, Larsdale had this to say, “Liberal rhetoric.” When asked if it was true that there were only 25 individuals with voting rights in BBC-Viacom Larsdale replied, “That's simply not true. Unfortunately we can't provide the news with exact financial details of our company since IAFAA but rest assured this is legitimate.”

    * IAFAA refers, of course, to the International Anti-Fiscal Abuse Act, which enables multinational corporations to hide financial information regarding their assets from individuals and government entities world wide. IAFAA comes in the wake of numerous witch hunts in the United States and Singapore to root out corporate corruption. The IAFAA ensures that such witch hunts will never happen again. The U.S. Supreme Court has already ruled such financial investigations unconstitutional without federal oversight.

     

    SO CAL and NO CAL

    * The Oakland Bridge phenomenon creating economic tensions between both sides. Businesses throughout the once-unified state continue to decline. Technological advancement as a whole seems a thing of the past. Without the protection of corporate arcologies, a number of viral outbreaks have plagued the inhabitants of California.

    * To the contrary our undercover sources on the Bridge report that civilization is thriving (albeit with a lower quality of life than elsewhere in the world).

    * Due to recent state statutes, California's state government has been dissolved. This comes as no shock to those that were expecting such years ago in the wake of multiple instances of corruption throughout the state.

    * There are no reports available from Night City.

     

    FREE STATES

    * New York's dome is falling! Death counts are at an all time high. 4,300 people have died directly or indirectly from the dome's disintegration: city officials have been trying to safely dismantle the dome for years now. But opposition from corporations continue.

    * Biotech company Osaka-Pfizer Pharma, GmB claims that they can actually fix the dome and that such reconstruction is essential to the peace and security of those within New York. They claim that by breaking down the dome a number of hazardous particles will undeniably spread throughout the city. In a vid conference with ex-CDC director William Winchester, an consensus was reached that the dome should stay in order to filter possible viruses and diseases from the outside world from coming into Manhattan.

    * Osaka-Pfizer also is pleased to release their new line of Synapse Neurococktails. In related news they've opened a new laboratory in Brooklyn, New York where they'll be developing new neurotransmitter supplements for every day consumption.

    * Osaka-Star Media, a private adult entertainment firm, is offering $500,000 for any information that leads to the capture of individuals that might have penetrated their network late last night. While the intrusion was not deep they claim that such actions should not be condoned, “Cyberterrorists are a threat to our city's safety and security and this is a fine example why,” said CEO Richard Rei from his office in Scarsdale, “We might sell adult entertainment but we're still a legitimate company. To emphasize this point we're asking the public to assist us in the capture of these Cyberspace Cowboys. The reward is very real.”

    * Osaka-Star Media and Osaka-Pfizer's parent company The Osaka Corporation declines to respond but offers their full support.

     

    Off World

    * Russian, Chinese and American corporations have resumed constructing weapons platforms on a number of space stations. More on this in the coming weeks.

     

    Other Reports of Record Sales

    * Maas Neotek, Osaka, Hosaka, Arasaka, Osaka-Star Media, Osaka-Pfizer, Biotechnica, Infocomp, Kendachi, Microtech, Militech, Orbital Air, Petrochem, World News Service (Wire), WorldSat, and Zetatech... the list goes on.

     

    NEWS NEWS NEWS NEWS NEWS NEWS NEWS

  5. They should be posting any day now... Eraser says he's interested and someone else from the forums messaged me. I think they just need pestering. From what I know I think Eraser even has a character planned out. If anyone is reading this: we'd love players! We're very social and fun... or something corny.

     

    As for Synapse, yes, I can totally see it messing with the body if someone is on any kind of medicine (hormonal treatment especially given the targeting of similar receptors in the brain). Also it is very addictive... was thinking of a whole Brave New World sort of thing, cure for society's ills through chemicals. Like anti-depressants now mixed with the addictiveness of McDonalds fast food. But worse.

     

    Anything you can offer for that whole angle would be helpful as always.

  6. What is the street traffic like? Is it heavily auto's, foot traffic or bikes and such. I assume that a pedicab can get around New York fairly well, all be it a tad slower at times depending on the amount of large death machines(cars and trucks). I assume the air that is breathed is not that healthy, right?

    Very heavy traffic and even more pedestrians running to go everywhere. So I'm guessing the cab would be hell to operate. Very slow but for someone that knows the ropes they could try to bypass the hustle and bustle with side streets, curb tactics and other such nuances. Pollution killer especially with a part dome.

  7. Hey all,

     

    This is for the game previously called "Night Run" on Data Fortress 2020. The game is still very active and we'd love for you to join us!

     

    What am I writing all this for? Simply put: I want to write a story with you! A story centric forum based free form Cyberpunk RPG for mature players.

     

    I've GM'd a lot of RP games so when thinking of something to do I decided against using the standard format seen elsewhere. I really want this game to be a free form game and I absolutely don't want you – the player – to feel limited in any way. Sure I'll try to come up with storylines and fun things to play but I really don't want players/friends to be bogged down with character sheets and histories. I'll assume – if you decide to play in these threads – that you have a character in mind, if you need help making one feel free to PM me and I'll try to help. If you want to post character sheets or share ideas, please do!

     

    That said I'm not an RP expert nor do I claim to be. All I ask is that if you take part in any threat that I'm in, you keep everything cool. No over powered characters. Nothing like that. Rich corporate types are fine... if you want to incorporate it into some sort of story, contact me and we can all work in it together. Novelists are welcome but if you want to make small posts, feel free. Remember description helps us all envision the world... be creative. Want to assume a bit of another players reply in making your own detailed post? You can if your post makes sense. Writing and storytelling are most important here.

     

    This game has been split into "OOC" threads and "IC" threads. Little to no OOC communication should occur in the In Character threads. Normal RP rules do apply, no using OOC information in character.

     

    Unlike a lot of the other forum based Cyberpunk games around I'm really interested in getting one that runs itself. Even if there is no storyline present, I want you – the player – to feel at home to create a character and jump right in and chat with others at locations mentioned in game.

     

    Ok so right now you might be thinking, “Well okay scout, what kind of setting do you have in mind?” I'm thinking of one which can span multiple cities during a 2020 time period. Gibson's Sprawl Trilogy, Bridge Trilogy and short stories are a great reference. Anything works but this game is very Gibson-esque. Never read any William Gibson? Consider the RP system Cyberpunk 2020 or, hell, even Shadowrun (if you want to play an orc you might be considered weird though!). If you know exactly what I'm talking about, cool. You don't need to know the Chrome books inside and out but do have some knowledge of the technology involved.

     

    Already I've said too much. Feel free to PM me with questions or concerns.

     

    Common issues...

     

    Bioware and technology:

     

    I should probably state that most bioware/implants/programs from the Chrome books can be available to starting characters so long as it truly reasonably fits the character (and the character's economic status and access to such technology). Essentially as long as you could explain it, should I ask you to, the reason for having such technology should be understandable.

     

    Another element of game play should be the loss of humanity. In Cyberpunk 2020 this concept has a different name but for all intents and purposes one should just consider that the more you modify the characters human body the less human he or she will become. While I don't expect anything drastic for something as mundane as a single cyberarm, biomods dealing with data chips in the brain might alter the individuals humanity.

     

    Similarly characters can buy such items through made up NPCs in game or from other characters and have them installed accordingly. You need not have any special scenes to do this, just describe what is happening and who they are receiving it from in your regular posts. The character's actual money won't - chances are - be recorded by me but you shouldn't spend it liberally. Remember to treat your character as realistic as possible.

     

    Abusing this free form system won't be good but I don't imagine that happening. Similarly if there are biomod upgrades related to appearance that you really think your character would have, do it. But something like Ikons on standard hooker or jockey isn't very realistic or likely. Along the same lines abuse would be considered full body replacements for no apparent reason, with no permission or notice before hand in which the character is super powered and can take down corporate shock troops easily.

     

    *+* As for Special Abilities, as long as they make some sense for the type of character it is they are in. Since reasonably speaking a jockey could potentially be good at combat limiting them in such a way wouldn't be good. Since we aren't really rolling (or at least we aren't rolling for actions any time soon), the question becomes whether or not it is reasonable for the character to possess such abilities. A military trained jockey might have a great reason for possessing both types of skill sets. Only I'd argue perhaps that they aren't as good at either one as much as they would be if they only focused in one.

    ...

    *+* The characters which I'd say are out of the question are NPC only entities, constructs by and large, AI, powerful kingpins without permission before hand and other such oddities. This is really more of an exception than a rule: solo characters, netrunners, social and corporate types, nomads, all of that is allowed.

     

    Humanity rules associated with loading up on too much machine-wear applies.

     

    That said I will try to incorporate all characters in big storylines and exclude characters if the player doesn't want his or her character involved in that particular story. Technically creating a large world instead of running small quests/runs as frequently (I'm hoping players will try to come up with storylines as well but it isn't required). So, as an example, if 4 characters are buddies in game and work together and are taking down a megacorp and two players don't really want to get involved, they don't have to (I realize how abstract this is sounding and I apologize).

     

    Essentially my goal is to have the game more like cooperative story writing rather than fast game play. Characters do what they want to do as long as it doesn't hurt other characters, doesn't provoke NPC police/private corporate security, and abides by all repercussions that would normally occur. But that isn't to say people will be sitting around not doing anything... I will try to move things along by using NPCs as plot movers. :P

     

    *+* As for UI, yes, the use of UI rules and systems along with the Cyberpunk 2020 core book and related supplements can be used (similarly the biotech offered by them may be used as well). Any material outside of that might need prior approval (only because I might not have read the material in question and have questions about an individuals character).

     

    *+* People can start posting... right away. You can send me some background on your character or just jump in if you feel comfortable. I realize the standard "meet at a bar" might sound cliche but really it is all up to you. If your character wouldn't interact with other characters presently in the game (not really the case here since there are no other characters beside mine in the game), then they need not interact with them.

     

    The reason why my rules aren't that strict is because I really want players to feel like they can create the characters they want to portray. Story matters not characters or stats but the characters must be reasonable.

     

    Hope this explains otherwise obscure element to free form play!

  8. Iron Crown Enterprises (ICE) Cyberspace was pretty cool.

     

    Complex as hell in parts, with awesome critical tables. Later time period (2090) and it did have a wealth of material available (published).

     

    Percentile based system.

     

    One of the many sources I cribbed stuff from in the early days.

    I've picked up a copy of ICE's Cyberspace from a friend and I have to admit there are a few elements that I find a bit strange. One of those happens to be the profession system: I find a lot of cross over, for instance, between the Net Junkie and the Jockey and find myself wondering why one relies on general skills and the other on decking skills. To explain this they maintain that a Jockey doesn't necessarily know the machinery he or she uses, she just uses it and understands technology through a general skill-set.

     

    Totally different from 2020 and I find myself liking 2020's system more in this regard (but that's it, the rest I considered on its own merits). But I really dig how it mentions that GMs can use different names for the professions and that nothing is set in stone (the common White Wolf-esque line). Those professions given are more general guidelines. That's how I like to play when running a freeform game... an aspect I really like.

     

    All in all it seems to be a great game onto itself with its own intricate Cyberpunk rules. It provides some great Cyberpunk related stories/history for the CP world. Thanks for sharing, very cool alternative to the standard Cyberpunk 2020 game.

     

    the major setting difference is that you play kids with super powers....

    Totally different type of setting from Cyberpunk 2020 then, but might have its own value. The gangs sound fun... I'd love to pick up a copy just based on that alone.

     

    As for arcs... yeah. Different system, I know, but pg. 63 of ICE's Cyberspace has some material covering arcologies but it is pretty standard. Wish they had more too. When I describe them in RPs I make them from scratch usually, research a lot about architectural environments before hand, that sort of thing.

  9. Thanks very much! Looks like a great resource for dling rpg material.

     

    Off on a similar topic of fantasy mixed with Cyberpunk...

     

    If anyone likes Vampire: The Masquerade and Cyberpunk do not pick up that cross over source book. It is horrible (although the fact that it is a cross over between two distinctly different RPGs should tell you as much). Shadowrun works or player made conversions to 2020 exist on the net, whereby the actual "goth" counterculture elements present in the Sprawl Trilogy are described in more fantastic terms, actual vampires to some degree. I won't really go into it here as I'm not a big fan of mixing fantasy with my Cyberpunk (outside of Shadowrun that is), but thought people should know such exists out there if interested.

     

    Edit: The text previously mentioned is World of Future Darkness. It appears as though the copyright might have lapsed (article was from White Wolf Magazine #36). I'll gladly send it to anyone if they are interested but again I've had no use for it. It could have been done a lot better.

  10. Cool, I've never heard of this CyberGeneration setting.

     

    The game is really awesome, looks like it fits more with Gibson-esque setting. Is that so? Are there major setting differences? Wish I could buy it but that PDF bundle is so expensive. :)

  11. Okay the way I handle things like that is I typically involve the police in game if I can. NPC them coming to the scene if it is such an instance (like where a character starts firing off a gun somewhere). Then, yeah, you can pretty much do whatever you want from lock up the character, take the character over (as an extreme measure), or hurt 'em really bad.

     

    If they got cyberware have it malfunction. Totally your right to do as a GM. It really does depend on the circumstances but I can totally see throwing them up against a group of gang sorts. I agree with the whole idea that you don't want anything too OP equipment wise because you don't want the character to take it (hat off to Wisdom for that). But the sniper shot might be a little unfair even with a warning and I'd never do it.

     

    Plus it is kind of unreasonable... unless they are in the middle of a warzone... story-wise.

     

    See that's the problem when playing with people you don't know too well also. I play with people that actually want their characters to live and typically don't test my patience. If a player is bored like Doom said the best approach is to stop playing for a while. If it is a new player that is just flexing their muscle a bit lead by example not by your awe inspiring powers as a GM. I can easily pull out a dragon... hell, I could reach over and stab the player in the heart... it just isn't worth it.

     

    The goal isn't to flex GM muscle at the player, it's to get them to be a contributing, productive member of the game. I personally don't feel this is best achieved through attacking their character, no matter how cleverly it's done. To my mind, that is still thinking like a player and not leveraging the GM position fully.
    That's how I always thought of it too. Beside I really don't want to kill a player's character... that just ruins the fun.

     

    I suppose it is a style of play question as well. I'm big on stories and not so big on random occurrences. D&D games my friends have run have all been, “You go into the abandoned inn and... SHIT! You notice a giant lamb with vampire fangs coming to attack you.” …Yeah, not happening with me. Random dragon scenarios don't do it for me either.

     

    Facing frequent attacks that aren't tied directly to the story teaches players to make characters as irrationally combat-ready as possible...
    I couldn't have said it better.
  12. Heya Drifter,

     

    Your name is absolutely awesome. And really great to have someone else from the Cyberpunk LARPing cadre. A small elite few from around the world that keeps the tradition alive.

     

    I've never read the Mirrorshades Anthology. Sounds interesting though. Our library has very few sci-fi books but I might just have to order it. My most recent read was the Wasteland Anthology comprised of a bunch of post-apocalyptic works. There's no real CP in it but it's still interesting. Will be tapping into Spook Country or Pattern Rec next.

     

    I'll stop talking now. Welcome to the forums!!!

  13. I remember ages back, when every one was excited for v3. Long live v2, well till there is something better.

    You have checked out Wisdom's -Interlock Unlimited- rules reboot right? B)

    I'm still drooling over your netrunners system :)

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